本篇回應
HCR 認為 含有正確訊息
引用自 HCR 查核回應
醫學研究證實,【精神分裂症 和 腦控、電子精神控制】相關的微波聽覺效應的醫學生理機制,【正好完全相反】
也就是,受電子攻擊者絕不是,也不可能會是精神分裂症(常見也幾乎是唯一被引導為此症來迫害,被害妄想、幻聽、幻覺),
已有醫學根據。醫學學理上就是完全不相干甚至相反的。
可見這些年來多少正是OSEH的無辜者「被精神病」當手段「從社會上做掉」了。
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先電子騷擾,再迫害進經神病機構,吃藥造成不可逆地吃壞腦子?
精神分裂症藥,是所有常見的精神科藥物裡最毒且最不可逆作用且確定會損傷大腦的。這些都是這類犯罪手法行之有年,有規劃過的。
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這篇,和【美國防預算書、美國防授權法案 (National Defense Authorization Act)】
明確列出,DEW指向性能源武器 的預算編列金額、組織狀況、研發概況,
【這堪稱同為這系列最重要的兩篇】好好珍惜善用。
這堪稱同為這系列,幫忙找出及張貼過來的資料裡,目前最重要的兩篇。好好珍惜善用。
1.解決有這種武器的點,2.解決會「被這種加害方式搞出社會、社會隔離、社會死亡」的點。
其它也是很重要的線索、細節,【也許也有人提供】而散落、放在網路的一些角落,好好網路尋找善用。
 
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腦控、電子精神控制裡、OSEH裡的EH,電子騷擾武器,最大宗的就是DEW,指向性能源武器,
而它又常包含三大項:光學(常見的雷射也算、有受害者會被不可見光影響視覺)、聲學(次聲波),電磁學(微波),
美國說他們有將敵軍癱瘓在灘頭的武器(號稱癱瘓,或暫時停止生命跡象,其實大功率足以直接結束生命,美國號稱得費時幾分鐘內,對岸解放軍的軍武專家比較老實,曾採訪時直說:那類武器,一兩分鐘內,甚至只要幾秒鐘內)、
抗暴,阻止抗議人潮常聽說的聲波鎮暴車(已有國家購買或使用過)、俄國的殭屍槍(中小型的DEW)
這些都算是DEW。美國防授權法案都說有,編列多年了,
台灣自己說沒有?說人類科技不可能?
且還是常駐霸占盤據cofacts的特定幾個編輯,上來聯手踩低文章?
https://www.facebook.com/groups/cofacts/posts/3294888530742953/
看看裡面有多少常駐的cofacts查核志工,專門上來踩這系列的查核文,並給自己同道隨意說不可能的文點讚?
都是同一幫人。
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精神分裂症的幻聽幻覺,和腦控、電子精神控制相關的微波聽覺效應(又稱弗雷效應,是指由脈沖或調變微波作用於人腦時,會產生出可聽喀嚓聲的現象或聲音或相關頭痛及副作用)的醫學生理機制,正好完全相反。

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https://www.reddit.com/r/TargetedIndividuals/comments/97mab8/j_symptoms_microwave_auditory_effect_mental/

[J] [症狀:微波聽覺效應] [心理健康:生物標誌物] 聽覺幻覺和精神分裂症是由紋狀體多巴胺過多引起的。而微波聽覺效應會降低多巴胺。
與紋狀體多巴胺相關的幻覺的知覺推理機制(2018)

https://www.ncbi.nlm.nih.gov/pubmed/29398218

未接受藥物治療的精神分裂症患者紋狀體連接功能地形的多巴胺相關破壞(2016)

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5310843/

沒有過多紋狀體多巴胺的人不會得到抗精神病藥物的幫助。

抗精神病藥物在精神分裂症發育大鼠模型中快速誘導多巴胺神經元去極化阻滯(2012)

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3167216/

精神分裂症的突觸前多巴胺合成能力和抗精神病藥物治療和無藥物條件下的紋狀體血流變化。(2017)

https://www.ncbi.nlm.nih.gov/pubmed/28387222

然而,受到電磁場 (EMF) 攻擊的 TI 的多巴胺含量較低。微波聽覺效應,也稱為聲音到頭骨 (V2K) 會影響大腦。

[WIKI] 神經遞質:多巴胺缺乏症

https://www.reddit.com/r/Electromagnetics/comments/62vghd/wiki_neurotransmitters_dopamine_deficiency/

多巴胺過多的人可能會自然地降低多巴胺。向整體心理學家或自然療法醫師諮詢神經遞質測試。

聽到聲音並且多巴胺過多的人沒有微波聽覺效應。他們不是 TI。
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https://www.reddit.com/r/TargetedIndividuals/comments/97mab8/j_symptoms_microwave_auditory_effect_mental/


[J] [Symptoms: Microwave Auditory Effect] [Mental Health: Biomarkers] Auditory verbal hallucinations and schizophrenia are caused by excessive striatal dopamine. Whereas, microwave auditory effect lowers dopamine.
A perceptual inference mechanism for hallucinations linked to striatal dopamine (2018)

https://www.ncbi.nlm.nih.gov/pubmed/29398218

Dopamine-related disruption of functional topography of striatal connections in unmedicated patients with schizophrenia (2016)

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5310843/

People who do not have excessive striatal dopamine will not be helped by antipsychotic drugs.

Antipsychotic drugs rapidly induce dopamine neuron depolarization block in a developmental rat model of schizophrenia (2012)

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3167216/

Presynaptic Dopamine Synthesis Capacity in Schizophrenia and Striatal Blood Flow Change During Antipsychotic Treatment and Medication-Free Conditions. (2017)

https://www.ncbi.nlm.nih.gov/pubmed/28387222

Whereas, TIs who are attacked by electromagnetic fields (EMF) have low dopamine. Microwave auditory effect, also known as voice to skull (V2K) zaps the brain.

[WIKI] Neurotransmitter: Dopamine Deficiency

https://www.reddit.com/r/Electromagnetics/comments/62vghd/wiki_neurotransmitters_dopamine_deficiency/

People who do have excessive dopamine may be able to lower their dopamine naturally. Ask a holistic psychologist or naturopath for a neurotransmitter test.

People who hear voices and have excessive dopamine, do not have microwave auditory effect. They are not TIs.

資料佐證

https://www.reddit.com/r/TargetedIndividuals/comments/97mab8/j_symptoms_microwave_auditory_effect_mental/
https://www.facebook.com/groups/cofacts/posts/3294888530742953/

A Perceptual Inference Mechanism for Hallucinations Linked to Striatal Dopamine - PubMed

Hallucinations, a cardinal feature of psychotic disorders such as schizophrenia, are known to depend on excessive striatal dopamine. However, an underlying cognitive mechanism linking dopamine dysregu

https://www.ncbi.nlm.nih.gov/pubmed/29398218

Dopamine-related disruption of functional topography of striatal connections in unmedicated patients with schizophrenia

Despite the well-established role of striatal dopamine in psychosis, current views generally agree that cortical dysfunction is likely necessary for the emergence of psychotic symptoms. The topographi

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5310843/

Antipsychotic Drugs Rapidly Induce Dopamine Neuron Depolarization Block in a Developmental Rat Model of Schizophrenia

Repeated administration of antipsychotic drugs to normal rats has been shown to induce a state of dopamine neuron inactivation known as depolarization block, which correlates with the ability of the d

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3167216/

Presynaptic Dopamine Synthesis Capacity in Schizophrenia and Striatal Blood Flow Change During Antipsychotic Treatment and Medication-Free Conditions - PubMed

Standard-of-care biological treatment of schizophrenia remains dependent upon antipsychotic medications, which demonstrate D<sub>2</sub> receptor affinity and elicit variable, partial clinical respons

https://www.ncbi.nlm.nih.gov/pubmed/28387222

A Perceptual Inference Mechanism for Hallucinations Linked to Striatal Dopamine - PubMed

Hallucinations, a cardinal feature of psychotic disorders such as schizophrenia, are known to depend on excessive striatal dopamine. However, an underlying cognitive mechanism linking dopamine dysregu

https://www.ncbi.nlm.nih.gov/pubmed/29398218

Dopamine-related disruption of functional topography of striatal connections in unmedicated patients with schizophrenia

Despite the well-established role of striatal dopamine in psychosis, current views generally agree that cortical dysfunction is likely necessary for the emergence of psychotic symptoms. The topographi

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5310843/

Antipsychotic Drugs Rapidly Induce Dopamine Neuron Depolarization Block in a Developmental Rat Model of Schizophrenia

Repeated administration of antipsychotic drugs to normal rats has been shown to induce a state of dopamine neuron inactivation known as depolarization block, which correlates with the ability of the d

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3167216/

Presynaptic Dopamine Synthesis Capacity in Schizophrenia and Striatal Blood Flow Change During Antipsychotic Treatment and Medication-Free Conditions - PubMed

Standard-of-care biological treatment of schizophrenia remains dependent upon antipsychotic medications, which demonstrate D<sub>2</sub> receptor affinity and elicit variable, partial clinical respons

https://www.ncbi.nlm.nih.gov/pubmed/28387222

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